Assistant Professor Joint Department of Biomedical Engineering at North Carolina State University and the University of North Carolina at Chapel Hill Raleigh, NC
Skin is constantly exposed to mechanical forces. Wounding significantly alters this environment leading to a disruption in mechanical homeostasis and alterations in mechanotransduction. These mechanical cues not only alter cellular function but also alter extracellular matrix stiffness and organization and are powerful mediators of both normal and abnormal healing. This session will explore how the mechanical environment controls wound healing from inflammation to remodeling and will discuss recent advances in therapies aimed at controlling the mechanical environment of healing wounds.